Skip to main navigation Skip to search Skip to main content

Decreased ability to manage increases in reactive oxygen species may underlie susceptibility to arrhythmias in mice lacking Scn1b

  • Jessa L. Aldridge
  • , Emily Davis Alexander
  • , Allison A. Franklin
  • , Chad R. Frasier
  • Quillen College of Medicine

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Pages (from-to)H723-H732
JournalAmerican Journal of Physiology - Heart and Circulatory Physiology
Volume327
Issue number4
DOIs
StatePublished - Oct 2024

ASJC Scopus Subject Areas

  • Physiology
  • Cardiology and Cardiovascular Medicine
  • Physiology (medical)

Keywords

  • arrhythmia
  • glutathione
  • reactive oxygen species
  • Scn1b
  • SUDEP

Cite this